Ask Lishui: What’s the Reason for Hypercholesterolaemia Associated With Nephrotic Syndrome?

"Christ in the Garden of Gethsemane" by Robert Walter Weir (courtesy metmuseum.org)
"Christ in the Garden of Gethsemane" by Robert Walter Weir (courtesy metmuseum.org)

From Cara Liu via Quora.com:

"What's the reason for hypercholesterolaemia associated with nephrotic syndrome?"

Symptoms Versus Signs

Hypercholesterolaemia is a sign and nephrotic syndrome - like all syndromes - is, by definition, a collection of symptoms.

The most prevalent symptom is persistent weight gain, not because of overeating (the appetite is actually poor), but because of extreme water retention in the chest, belly, and face areas.

Another symptom in this syndrome is fatigue.

Symptoms are discomforts that we experience. Signs are picked up in tests, but often go completely unnoticed otherwise.

Every symptom is an appropriate and meaningful biological response to stress. The type of stress determines the type of stress response and therefore the type of symptom.

(I give a basic overview of this approach to healing in my free e-book)

During the active period of the stress, one symptom (or set of symptoms) will develop, with attendant signs. When we resolve the stress, our metabolism (and the affected body system) will create a different symptom in the healing phase, with its own attendant signs.

Hypercholesterolaemia

Hypercholesterolaemia, a form of hyperlipidemia/hyperlipoproteinemia also called "dyslipidemia" is, in plain English, a high level of cholesterol in the bloodstream.

This is a sign, not a symptom - the person would not know that they have high cholesterol/fat levels unless you do a blood test.

The high blood cholesterol doesn't feel bad or uncomfortable ...but it is a sign that a certain symptom is developing.

High cholesterol/fat is most often associated with the healing phase after a stress affecting the coronary arteries. The coronary arteries are affected by a territorial loss conflict. Therefore, the hypercholesterolaemia is a sign of a resolved (or resolving) territorial loss conflict.

Assuming a coronary artery conflict due to territorial loss, the biological purpose for hypercholesterolaemia is to repair the linings of the coronary arteries, which were deteriorated during the active territorial loss conflict.

The biological reason for deterioration of the linings of the coronary arteries was to provide greater blood flow to the heart, in order to better equip the person to fight and get their territory back ("territory" can include a sexual partner, a status position, or property).

During the active phase of this territorial loss conflict, the person would have experienced depression and pain in the left side of the chest. In the healing phase, the person has a general sense of wellbeing and relaxation, may lack sensation in the left side of the chest, and will develop hypercholesterolaemia. 

If the territorial loss conflict is properly resolved and healing progresses without complication, the person will have a heart attack about 6 weeks in. (Non-fatal heart attacks often happen while we are sleeping, at about 3 a.m.)

Nephrotic Syndrome

A syndrome is, by definition, a collection of symptoms. In nephrotic syndrome, the primary symptom is swelling/fluid retention. Other symptoms are fatigue and loss of appetite, and changes to the quality and frequency of urination. 

Signs of nephrotic syndrome include protein in the urine and glomerulosclerosis, detected through medical tests, but generally unnoticed by the person who has this condition.

Each of these signs and symptoms has an explanation in the context of the 5 Natural Laws of Healing.

The swelling and fluid retention are the result of an active phase conflict (an active stress) to which we biologically respond by narrowing our kidney collecting tubules, causing a retention of fluid in the body.

The kidney collecting tubules become narrower by way of microscopic increases in tissues (secretory type compact cauliflower-like or resorptive type flat-growing adenocarcinoma on the inner linings of the kidney collecting tubules, between the renal calyces and the glomerular kidney parenchyma).

The kidney collecting tubules narrow during an active existence/refugee/ abandonment conflict.

The biological purpose of this symptom is to save water in order to attain a longer survival time. Its purpose is to literally protect the tissues of the body with a cushion of water and to carry necessary resources of survival close to the body.

(Consider a fish that accidentally gets beached: it will attempt to bring the ocean with it. Similarly, a traveller lost and disoriented in the desert will also retain as much water as possible)

Appetite loss is normal during any active stress response (active conflict phase).

Tiredness is a normal part of healing, but fatigue is more likely an active stress response; in this case, to protect the body by limiting expenditure of energy.

(Fatigue may also be caused by yet another conflict: an active self-devaluation conflict resulting in anemia and adrenal fatigue.)

During the active phase of the existence/refugee/abandonment conflict, urine retention ("uremia") is not "kidney failure;" it is actually the body's storage of uremic substances as an emergency protein storage mechanism (the nitrogen cycle is an important part of the metabolic breakdown and use of protein).

Conventional medicine sees urea breaking down into protein and eliminated through the urine. What has not been understood is that we are able to recycle urea into biologically-available protein (without peeing it out). So, in time of urgent protein need, we retain urea in the blood, which we call "uremia," and have incorrectly regarded as a disease symptom instead of a sign of a protective mechanism.

The Connection Between Hypercholesterolaemia and Nephrotic Syndrome

Now I would like to introduce an entirely different diagnosis: the kidney collecting tubule syndrome.

During the active phase of an existence/refugee/abandonment conflict, there is cell proliferation in the kidney collecting tubules resulting in fluid retention. This has enormous significance when it occurs together with the healing phase of a different conflict.

In combination with the healing phase of a different conflict, the fluid retention causes a large edema in the affected organ and associated area, which both intensifies and prolongs healing.

For example, healing-phase leukemia plus the active kidney collecting tubule conflict will become gout (which lasts much longer and is much more painful). Healing phase pleurisy plus the active kidney collecting tubule conflict will become pleural effusion (which can collapse the lungs). A healing phase runny nose plus the active kidney collecting tubule conflict will become sinusitis. A little extra healing phase fat plus the active kidney collecting tubule conflict will become cellulite. Healing conflicts in the associated brain area plus the active kidney collecting tubule conflict will become "brain tumours."

With the excess fluid pressure brought on by healing from one conflict in the presence of an active kidney collecting tubule conflict, we feel foggy, disoriented. We may develop divergent strabismus.

In the case of acute water retention in the whole organism, and especially in an organ that is healing, the water retention is biologically protective. The body still eliminates necessary uremic substances, even if the creatinine level increases to 12-14 mg%. This increase of creatinine (and of uremic acid, depending on what other healing phases may be occurring) provides a necessary extra availability of protein because we are able to recycle these substances into protein in a biological emergency.

In the case of healing of the territorial loss conflict, there has been angina during the active stress phase and there are various healing processes going on in this chest area after resolution of the conflict. This, combined with the active existence/refugee/abandonment conflict sets up the kidney collecting tubule syndrome and will create excessive fluid retention in the entire area around the healing tissues that had been called into play during the earlier (but now resolved) territorial loss conflict.

Healing will also be prolonged, and it is easy for the conflict to be re-triggered, which will interrupt healing. Thus, the combination can become a chronic illness.

Healing the Syndrome

It is absolute top priority to resolve the active existence/refugee/abandonment conflict.

During our healing from a serious conflict, we require extra rest and nutrition. The person who has this syndrome is trapped in "hanging healing," unable to fully resolve either the territorial loss conflict (which affects the coronary arteries) or the existence/refugee/abandonment conflict (which affects the kidney collecting tubules).

Biologically, he or she waits for safer conditions, much the way an animal will not go into labour until she finds a safe place to do so.

In this context, what has likely happened is that people with this diagnosis began healing in their chest area ...but then they felt unsupported and alone. Their healing symptoms made them feel that their physical existence was threatened.

Give an environment of unconditional support and assurance that their needs will be well met. Give a diet with plenty of quality protein. Then the person can begin to let go of their sense of feeling isolated and unsafe.

When this occurs, the linings of the kidney collecting tubules will immediately begin to slough off. In the presence of tuberculosis mycobacteria, we will see lots of protein in the urine, cloudy urine, etc. This is metabolic expulsion of no-longer-needed tissues.

It's a process!

Be well.

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